Anticancer treatment – why does it hurt so much and can we make it easier for patients?

3 May 2018, 18:30 - 19:30
Cambridge campus

Chris Parris

Inaugural lecture with Professor Chris Parris

In the UK there are over 360,000 new diagnoses of cancer every year and this number is increasing yearly.

During our lifetime one in two of us will develop cancer and require some form of treatment, which may be surgery, radiotherapy and chemotherapy (or all three) depending on the type and extent of cancer spread. Most cancer treatments kill cancer cells by attacking and destroying the DNA of the cancer cells so that they are unable to divide and grow.

However, normal non-cancer cells in the body are also killed by anticancer treatment and because of this patients experience unpleasant and painful side effects. Common side-effects include hair and weight loss, reduced appetite and increased infections due to suppression of the immune system. Cancer and normal cells can repair the damage to DNA during treatment and most cells have complicated DNA repair systems to reverse the effects of anticancer treatment. This lecture will explore how we can measure DNA repair in normal and cancer cells and use this information to develop individualised treatment to avoid side effects and make treatment more tolerable for cancer patients.

Professor Chris Parris arrived at Anglia Ruskin University in April 2017, where he is Head of the Department of Biomedical and Forensic Science, in the Faculty of Science & Technology. Previously, Chris was employed at Brunel University West London where he was Head of the Biosciences Division in the College of Health and Life Sciences.

Chris completed his first degree in Biology at Nottingham University and after a short period as a qualified Biomedical Scientist in the NHS, completed his PhD in the cell biology of cancer at University College London in 1989. Chris is an active researcher and has developed a thriving research group in the area of human DNA repair mechanisms and how inherited defects in DNA repair leads to extreme patient sensitivity to DNA damaging and increased cancer risk.

Event Details

3 May 2018, 18:30 - 19:30
Cambridge campus
Science Centre, SCI105

Please book online.